0:00

On the left,

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you have a study from September 4, 2018,

0:05

in a patient who had hepatorenal syndrome.

0:08

And what one sees on the images is

0:14

the abnormal signal intensity.

0:15

It's somewhat hazy in its appearance,

0:18

affecting the posterior temporal lobe and the

0:20

occipital lobes and the parietal lobes.

0:23

Let's just highlight that with our magic pen here.

0:28

So this is what I'm referring to,

0:29

is that sort of hazy,

0:31

bright signal intensity that's extending to the

0:34

subcortical white matter fibers,

0:38

associated with bilateral involvement

0:40

of the parietal lobes.

0:42

However,

0:43

this includes the involvement of the occipital

0:46

lobes as we scroll further inferiorly,

0:49

which is again demonstrated to extend all the

0:52

way to the periphery here of the white

0:55

matter of both occipital lobes.

0:59

This is the September 4 study.

1:02

On the right-hand side is the same patient,

1:05

and the study is from October 5th.

1:09

So, basically,

1:10

four and a half to five weeks later.

1:13

And as you can see,

1:16

there is complete resolution of the involvement

1:19

of the occipital lobes,

1:21

as well as the parietal lobes, with no residual,

1:25

no encephalomalacia,

1:27

ventricle size, the same.

1:30

Looks like it's back to normal.

1:32

And that is the typical course that we see with

1:34

posterior reversible encephalopathy syndrome,

1:37

as well as the reversible cerebral

1:41

vasoconstrictive syndrome.

1:43

So, this is the expected and hoped-for resolution

1:47

of PRES over the course of weeks,

1:50

once the underlying etiology for

1:54

PRES has been reversed.