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Training Collections
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Black Friday Save 30%Practice-focused training programs designed to help you gain experience in a specific subspecialty area.
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Black Friday Save 40%Unlock access to our full Course Library and all self-paced Fellowships.
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Get access to free live lectures, every week, from top radiologists.
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Dr. Resnick's MSK Conference
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Supplement your training program with case-based learning for residents, registrars, fellows, and more.
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Upskill in high growth, advanced imaging areas.
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49 topics, 3 hr. 16 min.
Inner Ear Preview
2 m.Inner Ear – Introduction
2 m.Anatomy of the Internal Auditory Canal (IAC)
8 m.Coronal Anatomy of the Inner Ear
4 m.Axial IAC Anatomy and Otospongiosis/Otosclerosis
6 m.Coronal IAC Anatomy and Facial Nerve Segments
6 m.MRI imaging techniques and cochlea aplasia
7 m.IAC Congenital Lesions & Syndromes - Summary
7 m.Cochlear Hypoplasia
8 m.Cochlear Nerve Deficiency, Pontine Tegmental Cap Dysplasia
5 m.Bilateral Cochlea Nerve Deficiency
5 m.Labyrinthine Dysplasia/Syndromes - Summary
10 m.Incomplete Partition Type 1
3 m.Incomplete Partition Type 2 – Summary
3 m.Bilateral Incomplete Partition Type 2
3 m.Mondini Malformation, Incomplete Partition Type II
2 m.Incomplete Partition Type II, Mondini Malformation, Semicircular Canal Abnormality
3 m.Vestibular Malformation
3 m.Enlarged Endolymphatic Sac
2 m.Incomplete Partition Type III – Summary
4 m.Down Syndrome – Summary
6 m.Down Syndrome, Semicircular Canal Deformity, Cochlear Aperture Stenosis
6 m.Down Syndrome, Aperture Stenosis
6 m.Cochlear Hypoplasia and Aperture Stenosis - Summary
4 m.Semicircular Canal (SCC) Dehiscence – Summary
4 m.Semicircular Canal (SCC) Dehiscence
3 m.Semicircular Canal (SCC) – Oblique Reformat
2 m.Inflammatory/Infectious Lesions of the Inner Ear - Summary
7 m.Labyrinthitis, Secondary to Otomastoiditis
3 m.Labyrinthine Fistula Mastoidectomy and Cochlea implant
3 m.Viral Labyrinthitis
3 m.Otospongiosis (Otosclerosis) - Summary
10 m.Bilateral Otospongiosis (Otosclerosis)
5 m.Bilateral Retrofenestral Otospongiosis
4 m.Bilateral Otospongiosis and SCC Dehiscence
3 m.Otospongiosis, Left Stapedectomy
3 m.Labyrinthitis Ossificans – Summary
11 m.Post Traumatic Labyrinthitis Ossificans
3 m.Labyrinthitis Ossificans, Cochlear Turn
2 m.Labyrinthitis Ossificans, Superior SCC
2 m.Unilateral Labyrinthine Ossificans
2 m.Petrous Apex Lesions
8 m.Right Cholesterol Granuloma
5 m.Intravestibular/Labyrinthine schwannoma
3 m.Labyrinthine Schwannoma
4 m.Left Side Labyrinthine/Vestibule Schwannoma
2 m.Endolymphatic Sac Tumor (ELST) – Summary
4 m.Endolymphatic Sac Tumor and VHL
4 m.Inner Ear Malignant Neoplasm and Trauma Closing Points
6 m.0:01
We just saw an example of incomplete partitian type
0:04
one where we really didn't have any evidence of a
0:08
mediolis in the cochlea, and it looked kind of bulbous.
0:12
And in that example, we saw a cystic cochlear
0:15
vestibular vestibule structure in
0:19
incomplete partitian type two.
0:22
We have a better development of the mediolis,
0:26
but it has incomplete spiralization of the cochlea.
0:30
And that is one of the findings that we
0:33
typically call Mondini malformation.
0:36
Now the eponym Mondini malformation has been fused
0:39
into the incomplete partitian type two.
0:43
Here is an example from Traboulsi's article in
0:47
Neurographics in which she identifies the abnormal
0:53
spiralization of the cochlea. We have a basal turn,
0:57
but the middle and apical turns are not developed, and
0:59
we don't see the mediolis associated with it.
1:03
Here again, basal turn of cochlea.
1:05
And then we have this blob for the second
1:08
and third turns of the cochlea.
1:11
And this is associated with enlargement
1:15
of the vestibular aqueducts.
1:16
So these are the vestibular aqueducts.
1:18
As you heard previously,
1:19
I said that we usually compare the caliber of the
1:23
vestibular aqueduct with the caliber
1:25
of the semicircular canals.
1:27
And when it is larger than the caliber
1:29
of the semicircular canals,
1:32
we consider it enlarged.
1:34
You can use one and a half millimeters as another of
1:38
the guides to an enlarged vestibular aqueduct.
1:42
Here is another example of incomplete partitian type
1:45
two associated with enlarged vestibular aqueduct.
1:49
This dilated structure here,
1:51
here's one of our semicircular canals, and you can
1:54
see that this is a portion of the cochlea.
1:56
We'd have to scroll down, but in this case,
1:59
the patient did have cochlear dysplasia as well.
Interactive Transcript
0:01
We just saw an example of incomplete partitian type
0:04
one where we really didn't have any evidence of a
0:08
mediolis in the cochlea, and it looked kind of bulbous.
0:12
And in that example, we saw a cystic cochlear
0:15
vestibular vestibule structure in
0:19
incomplete partitian type two.
0:22
We have a better development of the mediolis,
0:26
but it has incomplete spiralization of the cochlea.
0:30
And that is one of the findings that we
0:33
typically call Mondini malformation.
0:36
Now the eponym Mondini malformation has been fused
0:39
into the incomplete partitian type two.
0:43
Here is an example from Traboulsi's article in
0:47
Neurographics in which she identifies the abnormal
0:53
spiralization of the cochlea. We have a basal turn,
0:57
but the middle and apical turns are not developed, and
0:59
we don't see the mediolis associated with it.
1:03
Here again, basal turn of cochlea.
1:05
And then we have this blob for the second
1:08
and third turns of the cochlea.
1:11
And this is associated with enlargement
1:15
of the vestibular aqueducts.
1:16
So these are the vestibular aqueducts.
1:18
As you heard previously,
1:19
I said that we usually compare the caliber of the
1:23
vestibular aqueduct with the caliber
1:25
of the semicircular canals.
1:27
And when it is larger than the caliber
1:29
of the semicircular canals,
1:32
we consider it enlarged.
1:34
You can use one and a half millimeters as another of
1:38
the guides to an enlarged vestibular aqueduct.
1:42
Here is another example of incomplete partitian type
1:45
two associated with enlarged vestibular aqueduct.
1:49
This dilated structure here,
1:51
here's one of our semicircular canals, and you can
1:54
see that this is a portion of the cochlea.
1:56
We'd have to scroll down, but in this case,
1:59
the patient did have cochlear dysplasia as well.
Report
Description
Faculty
David M Yousem, MD, MBA
Professor of Radiology, Vice Chairman and Associate Dean
Johns Hopkins University
Tags
Temporal bone
Neuroradiology
Head and Neck
Congenital
CT
Brain
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