Interactive Transcript
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This is a 52-year-old woman, wife of a
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physician, who just slightly twists her
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ankle in a clogged shoe, stepping off the
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curb in front of a coffee shop, and the next
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day, she cannot bear weight on the foot.
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All kinds of diagnoses are postulated.
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All of her imaging is negative.
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She does not undergo bone scintigraphy,
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but she does undergo CT and X-ray and MRI.
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All of which are initially deemed normal.
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These are serially carried out over a
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period of time, and the lack of weight
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bearing and inability to do so persists.
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So approximately six weeks later,
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this MRI is performed again.
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And if you look at the sagittal, it's a low
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field study, if you look at the sagittal along
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the edges of the skeleton, the corners, the
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so-called shiny corner sign that you see in
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ankylosing spondylitis, is present in the foot.
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This appearance was not highlighted
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strong enough for the clinician.
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And the clinician decided that after a protracted
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period of non-weight-bearing, that the problem,
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which was localized, the pain was localized in
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the distribution of the medial plantar nerve
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that the patient needed to have her posterior
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tibial tendon taken off and re-implanted.
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And that indeed was done.
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At re-implantation, the bone was soft and
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difficult to harbor and fix the anchor.
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That tipped a group of physicians off to
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the correct diagnosis, which was complex
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regional pain syndrome type 2, in a nerve
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distribution most likely related to an injury
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of the tibial or medial plantar nerve that
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then germinated into diffuse type 1 RSD.
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This shiny corner appearance
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should have been the tip-off.
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This low signal intensity corner should have
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been the tip-off, but even more important is the
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prominence of the trabecula, which is a sign of
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osteopenia, and there was no other explanation
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for the patient's inability to bear weight.
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Let's see what happens in this condition
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when it goes unchecked on a CT.
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So on CT, unchecked.
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Massive osteopenia.
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And also hypersensitivity on the
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part of the patient to touch.
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Yet still, the patient's foot surgeon refused
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to allow the diagnosis of RSD, even though
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it had been questioned multiple times.
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Finally, the clinician was overridden.
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The patient was taken to an anesthesiologist.
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The patient underwent lumbar block, instantaneous
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improvement in both the vascularity of the
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foot. Decrease in pain, and all of this
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spotty, extensive osteopenia resolved.
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The patient now is normally ambulating.
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But this is how subtle reflex
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sympathetic dystrophy can be.
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It can start out with very subtle, shiny
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corners in the bone and nothing more.
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No soft tissue swelling, no effusion,
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and progress to massive osteopenia
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due to autonomic dysfunction.
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This is a proven case.
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